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cno enzo bml-ns105-0025  (Enzo Biochem)

 
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    Enzo Biochem cno enzo bml-ns105-0025
    a Acute novelty stress-induced Fos (purple) signals in the paraventricular hypothalamic nucleus (PVN) of <t>CNO-treated</t> NPY mcherry and NPY hM3Dq mice. Representative images ( a ) and quantitative data ( b ). Scale bar: 200 μm. In ( b ) Two-sided unpaired Student’s t test with Welch’s correction. c The increase of serum corticosterone levels caused by novelty stress in CNO-treated NPY mCherry and NPY hM3Dq mice. Two-sided unpaired Student’s t test. d Schematic of behavioral test design after 2-h acute novelty stress <t>with</t> <t>chemogenetic</t> manipulation. e Representative EPM traces of different mouse groups. The gray shades indicate the closed arms. f Percentages of open-arm time in the EPM. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. g Representative OFT traces of different mouse groups. The gray shades indicate the center of the arena. h Percentages of time, travel distance in the center area and total travel distance in OFT. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. Stressed mice were subjected to acute novelty stress for 2 hs prior to behavioral tests, and all stressed mice received CNO injections. Data are shown as mean ± SEM. ‘n’ refers to mice number. Source data are provided as a file.
    Cno Enzo Bml Ns105 0025, supplied by Enzo Biochem, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/cno+enzo/pmc11365948-489-13-17?v=Enzo+Biochem
    Average 90 stars, based on 1 article reviews
    cno enzo bml-ns105-0025 - by Bioz Stars, 2026-08
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    Images

    1) Product Images from "Feedforward inhibition of stress by brainstem neuropeptide Y neurons"

    Article Title: Feedforward inhibition of stress by brainstem neuropeptide Y neurons

    Journal: Nature Communications

    doi: 10.1038/s41467-024-51956-9

    a Acute novelty stress-induced Fos (purple) signals in the paraventricular hypothalamic nucleus (PVN) of CNO-treated NPY mcherry and NPY hM3Dq mice. Representative images ( a ) and quantitative data ( b ). Scale bar: 200 μm. In ( b ) Two-sided unpaired Student’s t test with Welch’s correction. c The increase of serum corticosterone levels caused by novelty stress in CNO-treated NPY mCherry and NPY hM3Dq mice. Two-sided unpaired Student’s t test. d Schematic of behavioral test design after 2-h acute novelty stress with chemogenetic manipulation. e Representative EPM traces of different mouse groups. The gray shades indicate the closed arms. f Percentages of open-arm time in the EPM. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. g Representative OFT traces of different mouse groups. The gray shades indicate the center of the arena. h Percentages of time, travel distance in the center area and total travel distance in OFT. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. Stressed mice were subjected to acute novelty stress for 2 hs prior to behavioral tests, and all stressed mice received CNO injections. Data are shown as mean ± SEM. ‘n’ refers to mice number. Source data are provided as a file.
    Figure Legend Snippet: a Acute novelty stress-induced Fos (purple) signals in the paraventricular hypothalamic nucleus (PVN) of CNO-treated NPY mcherry and NPY hM3Dq mice. Representative images ( a ) and quantitative data ( b ). Scale bar: 200 μm. In ( b ) Two-sided unpaired Student’s t test with Welch’s correction. c The increase of serum corticosterone levels caused by novelty stress in CNO-treated NPY mCherry and NPY hM3Dq mice. Two-sided unpaired Student’s t test. d Schematic of behavioral test design after 2-h acute novelty stress with chemogenetic manipulation. e Representative EPM traces of different mouse groups. The gray shades indicate the closed arms. f Percentages of open-arm time in the EPM. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. g Representative OFT traces of different mouse groups. The gray shades indicate the center of the arena. h Percentages of time, travel distance in the center area and total travel distance in OFT. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. Stressed mice were subjected to acute novelty stress for 2 hs prior to behavioral tests, and all stressed mice received CNO injections. Data are shown as mean ± SEM. ‘n’ refers to mice number. Source data are provided as a file.

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    a Acute novelty stress-induced Fos (purple) signals in the paraventricular hypothalamic nucleus (PVN) of <t>CNO-treated</t> NPY mcherry and NPY hM3Dq mice. Representative images ( a ) and quantitative data ( b ). Scale bar: 200 μm. In ( b ) Two-sided unpaired Student’s t test with Welch’s correction. c The increase of serum corticosterone levels caused by novelty stress in CNO-treated NPY mCherry and NPY hM3Dq mice. Two-sided unpaired Student’s t test. d Schematic of behavioral test design after 2-h acute novelty stress <t>with</t> <t>chemogenetic</t> manipulation. e Representative EPM traces of different mouse groups. The gray shades indicate the closed arms. f Percentages of open-arm time in the EPM. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. g Representative OFT traces of different mouse groups. The gray shades indicate the center of the arena. h Percentages of time, travel distance in the center area and total travel distance in OFT. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. Stressed mice were subjected to acute novelty stress for 2 hs prior to behavioral tests, and all stressed mice received CNO injections. Data are shown as mean ± SEM. ‘n’ refers to mice number. Source data are provided as a file.
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    a Acute novelty stress-induced Fos (purple) signals in the paraventricular hypothalamic nucleus (PVN) of <t>CNO-treated</t> NPY mcherry and NPY hM3Dq mice. Representative images ( a ) and quantitative data ( b ). Scale bar: 200 μm. In ( b ) Two-sided unpaired Student’s t test with Welch’s correction. c The increase of serum corticosterone levels caused by novelty stress in CNO-treated NPY mCherry and NPY hM3Dq mice. Two-sided unpaired Student’s t test. d Schematic of behavioral test design after 2-h acute novelty stress <t>with</t> <t>chemogenetic</t> manipulation. e Representative EPM traces of different mouse groups. The gray shades indicate the closed arms. f Percentages of open-arm time in the EPM. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. g Representative OFT traces of different mouse groups. The gray shades indicate the center of the arena. h Percentages of time, travel distance in the center area and total travel distance in OFT. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. Stressed mice were subjected to acute novelty stress for 2 hs prior to behavioral tests, and all stressed mice received CNO injections. Data are shown as mean ± SEM. ‘n’ refers to mice number. Source data are provided as a file.
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    Image Search Results


    a Acute novelty stress-induced Fos (purple) signals in the paraventricular hypothalamic nucleus (PVN) of CNO-treated NPY mcherry and NPY hM3Dq mice. Representative images ( a ) and quantitative data ( b ). Scale bar: 200 μm. In ( b ) Two-sided unpaired Student’s t test with Welch’s correction. c The increase of serum corticosterone levels caused by novelty stress in CNO-treated NPY mCherry and NPY hM3Dq mice. Two-sided unpaired Student’s t test. d Schematic of behavioral test design after 2-h acute novelty stress with chemogenetic manipulation. e Representative EPM traces of different mouse groups. The gray shades indicate the closed arms. f Percentages of open-arm time in the EPM. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. g Representative OFT traces of different mouse groups. The gray shades indicate the center of the arena. h Percentages of time, travel distance in the center area and total travel distance in OFT. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. Stressed mice were subjected to acute novelty stress for 2 hs prior to behavioral tests, and all stressed mice received CNO injections. Data are shown as mean ± SEM. ‘n’ refers to mice number. Source data are provided as a file.

    Journal: Nature Communications

    Article Title: Feedforward inhibition of stress by brainstem neuropeptide Y neurons

    doi: 10.1038/s41467-024-51956-9

    Figure Lengend Snippet: a Acute novelty stress-induced Fos (purple) signals in the paraventricular hypothalamic nucleus (PVN) of CNO-treated NPY mcherry and NPY hM3Dq mice. Representative images ( a ) and quantitative data ( b ). Scale bar: 200 μm. In ( b ) Two-sided unpaired Student’s t test with Welch’s correction. c The increase of serum corticosterone levels caused by novelty stress in CNO-treated NPY mCherry and NPY hM3Dq mice. Two-sided unpaired Student’s t test. d Schematic of behavioral test design after 2-h acute novelty stress with chemogenetic manipulation. e Representative EPM traces of different mouse groups. The gray shades indicate the closed arms. f Percentages of open-arm time in the EPM. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. g Representative OFT traces of different mouse groups. The gray shades indicate the center of the arena. h Percentages of time, travel distance in the center area and total travel distance in OFT. Ordinary one-way ANOVA with post-hoc Dunnett’s multiple comparisons test. Stressed mice were subjected to acute novelty stress for 2 hs prior to behavioral tests, and all stressed mice received CNO injections. Data are shown as mean ± SEM. ‘n’ refers to mice number. Source data are provided as a file.

    Article Snippet: For chemogenetic manipulations (except for manipulations during restraint stress), mice received injections of CNO (i.p., 2 mg/kg, ENZO, BML-NS105-0025) or an equal volume of saline half an hour prior to experiments.

    Techniques: